Dr. Gundry:
What if the real reason behind your anxiety, insomnia, and burnout isn’t in your mind, but it’s in your neurotransmitters? Welcome back to the Dr. Gundry Podcast. Today, we’re exploring a critical and shockingly overlooked piece of the health brain puzzle, GABA, the brain’s off switch. While dopamine and serotonin often dominate the headlines, a deficiency in GABA may be one of the culprits behind the skyrocketing issues of stress, depression, sleep disorders, and even dependence on anti-anxiety medications. My guest today is Dr. Scott Sherr, a board-certified internal medicine physician and one of the world’s leading voices in integrative and hyperbaric medicine. He’s on a mission to bring GABA deficiency into the spotlight and offer real practical solutions for restoring neurological balance, not with pills, but through advanced tools like fungaceuticals and precision lifestyle modifications.
We’ll be unpacking how GABA works, why supplementing with it often fails, and how you can truly support your brain’s natural ability to rest, reset, and recover. If you’re ready to opt out of the cycle of overstimulation and burnout and take back control of your brain, you’ve come to the right place. Stay tuned, we’ll be right back.
Now, let’s just dive right in. Because people are going, what the heck is GABA? What’s a GABA deficiency? Now, you’ve said that GABA deficiency is often the missing link in modern brain dysfunction, especially with anxiety, stress, and insomnia. Can you tell us about that?
Dr. Scott Sherr:
Sure. Yeah. GABA stands for gamma aminobutyric acid, and it’s the primary inhibitory neurotransmitter in your brain. Inhibitory means it calms things down. You have the gas pedal, which is glutamate, primarily. And you have the brake, which is GABA. Unfortunately, so many of us walking around right now are GABA deficient, and we don’t realize it. We have anxiety, insomnia, we have depression even. And it’s, not all of it, but it’s sort of like undertone that’s happening that clinicians and people don’t realize could be because this neurotransmitter is low or deficient. The thing about GABA, Dr. Gundry, is that it’s like the brakes of your brain. And so it’s like we have all these thoughts that are happening in our brain all the time. In fact, we have on average about 70,000 thoughts per day. That’s on average. And if we’re anxious and we’re stressed and we’re depressed, that number can go up to 120,000 thoughts per day.
And GABA is your break on all that. It works as something called an interneuron. It’s basically an area in the brain where you can have these way lay stations where GABA is trying to regulate the flow of information from your sensory system, from your motor system, and trying to bring it all together. If the GABA system is deficient and GABA neurons and GABA receptors are everywhere in the brain, if it’s deficient, then the brain cannot regulate itself. And so it’s overstimulated. It’s over-hyped. And that glutamate, that excitatory neurotransmitter is driving the show.
And between glutamate and GABA, this combination, these two neurotransmitters, that’s 80% of your brain’s neurotransmission. The superstar neurotransmitters, your norepinephrine, dopamine, serotonin, we all know about these, but what’s running the show is the balance between your glutamate and your GABA. And what’s interesting about that, is that in the brain it’s actually glutamate that gets converted into GABA with vitamin D6 and magnesium. So it’s always the balance between these two. And unfortunately, there’s so many things in modern life that are depleting GABA all the time for us.
Dr. Gundry:
I need to get me some GABA. How did we come to a point where… Obviously, we really should have a balance between the stimulatory factors and the off-switch factors.
Dr. Scott Sherr:
Right.
Dr. Gundry:
What’s happened that this is now unbalanced?
Dr. Scott Sherr:
Oh, it’s the hustle culture that you and I both know very well. It’s the on, on, on all the time. And not knowing how to turn off because that’s not what we’re programmed to do, especially Americans. And in medical school for me, we had shirts that said, “Sleep is for quitters.” You’ve heard sleep is something you do when you’re dead, for example. And this hustle culture, what it does is it drives our sympathetic nervous system. The sympathetic nervous system is the part of our nervous system that gives us fight or flight. It’s like, you ready, your action, go. Or you can run away if you need to. But the problem with that being all the time on is that it elevates a hormone called cortisol, which increases your blood sugar, affects your mitochondrial function, and also affects the enzymes that are responsible for that conversion over from that glutamate to GABA.
So you deplete the GABA and you can’t convert more of that glutamate over to GABA. And then, as a result of that, you are GABA deficient. In addition, of course there’s nutritional issues. There’s things that are required like vitamin B6 and magnesium that convert that glutamate over to GABA. So our poor diet, our lifestyle, our inflammation in our body. If we have leaky gut, as you know very well, this is going to cause systemic inflammation and going to affect those enzymes. And if you have a gut that doesn’t have the right types of bacteria in there too, that’s also going to be a detriment to the signaling of the GABA cascades that happen in the brain. Things like lactobacillus and bifidobacterium, for example, are very important microbiota that really help with the GABA signaling. So, we are overstressed, we are underslept, we have mitochondrial deficiency, we’re inflamed, and all of these things are causing a GABA deficiency.
And the problem is that you’re going to go to your doctor and you say, “I’m anxious doc, or I’m depressed,” and they’re going to give you an SSRI, for example. And we now know, and I’m sure you know this research too, Dr. Gundry, that there’s no evidence that depression is a serotonin deficiency. And this is the words that you would get from the drug companies back in the 90s and up until recently. But we do know that there is an indication that there may be a GABA deficiency going on, but doctors don’t know about it. I’m really excited to be able to present this information to your listeners because people need to know this. If you have anxiety, if you have depression, if you have insomnia, it could very well because that GABA system just needs better regulation.
Dr. Gundry:
Let me step back a second, because a lot of our listeners are going to say, “Well, wait a minute. I thought dopamine was the pay attention hormone. I’m dopamine stimulated. I’m playing a video game because I want that dopamine hit.” Is that not part of all this? Because everybody’s going, “Wait a minute, what happened to dopamine?”
Dr. Scott Sherr:
Well, dopamine is our reward drug. It’s our reward neurotransmitter. It’s a pleasure feeling. It gives you that feeling of satisfaction. And dopamine, of course, is a big part of what’s happening in modern life, because all of us are getting just slammed with information on a day-to-day basis. And the issue with dopamine fast and dopamine deficiency, this is all very true, and it’s all happening and taking time away. But what dopamine is really doing is stimulating that sympathetic nervous system again, so that’s the connection.
If your dopamine is up, you’re playing video games, you’re checking your phone every five seconds, you are giving yourself those dopamine hits. Those little sympathetic little bumps, little cortisol bumps that are happening every single time that you do that. And then over time, your GABA, your brain says, “Oh my God, there’s no more breaks I can give you because you’ve just depleted me. I don’t have enough, I’m not be able to convert over quickly enough. I don’t have the right nutrients anymore. I’m electrolyte deficient,” everything else. And so the dopamine part is really the sympathetic dominance part. So the goal really for all of us is to take the gas pedal off and turn the brakes back-
Dr. Scott Sherr:
The gas pedal off and turn the brakes back on. If we can do that and regulate our nervous system, that parasympathetic nervous system, that rest, digest, detoxify and recover nervous system better, then we’re going to perform better. We’re going to feel better. Because if we’re overstimulated, as many of you know that are listening, your brain doesn’t work as well. But if you can just calm down the nervous system just a little bit, then your brain starts turning back on, and you can function better, you’re energetically more coherent. That’s what the GABA system and regulating it really can do.
Dr. Gundry:
Should our listeners take away so far that they need to go to their doctor and have their GABA level measured?
Dr. Scott Sherr:
I love that you asked that, and the answer is no, unfortunately. Because GABA is only made, well, primarily made just in the brain. Now, there are some bacteria in the gut that I mentioned earlier that do some signaling across a nerve called the vagus nerve that helps with GABA production. But what you really need to think about is, number one, it’s a clinical diagnosis, and that’s hard, right? But as clinicians like you and I, we need to keep it on our radar when people are coming in. And those that are listening have to be thinking about it now too, so they can talk to their doctors.
What you can do, although you can’t check GABA directly, is you can check for the precursors that are responsible for making GABA in the body, and on some of the micronutrients that are responsible for that conversion over from glutamate into GABA. And so, the most common one you can check for is what’s called your glutamine level. Glutamine is an amino acid. It is the primary fuel of your small intestine, and it’s also very important for muscle growth, but it also converts into glutamate, and then from glutamate it gets converted into GABA. You can check your glutamine levels. I can promise you, if you have a leaky gut, you don’t have enough glutamine, because glutamine is responsible for trying to heal up that leaky gut. If you’re addressing the leaky gut, great, and that can be helpful. But if you have a leaky gut, you’re going to have a lot more challenges with this whole process overall. And so, you can check glutamine levels.
You can also check vitamin B6 and magnesium levels, because these can give you a sense of if you have enough magnesium or B6 to convert over. As I know you know, Dr. Gundry, 90% of US adults have magnesium deficiencies, so it is a big deal. Magnesium is also responsible for mitigating the glutamate signaling as well. That excitatory signaling that happens from glutamate gets mitigated when you have more magnesium around, it blocks those receptors. And so, magnesium is so important for so many things. I know you’ve spoken about this at length in various ways.
You can check magnesium, you can check B6, you can check copper, you can check zinc. These are all other things that are in and around the ecosystem that is responsible for making GABA. But in the end, it’s a clinical understanding of what this could be presenting as, and then doing some micronutrient testing, doing some gut testing, doing some amino acid testing. And then from there, it’s really trying to start working on lifestyle, diet, and then potentially supplementation to help support the system.
Dr. Gundry:
Recently, a health influencer who has been around a long time, who has a very visited website, is starting to say that, no, no, no, we’ve got it all wrong. This is a problem with the overproduction of serotonin. What say you?
Dr. Scott Sherr:
Well, I know who you’re talking about. And what I would say is this: our brains, our bodies, our minds are a beautiful ecosystem of networked balance. You don’t want to just demonize one neurotransmitter. I’m not demonizing glutamate, for example. I think it’s a fantastic one. It helps with memory, focus, attention, all these things. But in too much, it can cause excitatory symptoms and make people feel anxiety, insomnia, and depression, because they don’t have enough of that GABA. The same goes with serotonin. I think that there is reason to be upset about the world of serotonergic drugs, and SSRIs and SNRIs, because we know now that depression is not a serotonin deficiency. But my sense of things is that this is just something to think about more in a network fashion. We want to have balanced levels of serotonin, dopamine, and norepinephrine. We want to have balanced levels of GABA and glutamate. I don’t think we can really demonize one or the other.
But I would say that there is some evidence that serotonergic on high levels could be detrimental to mitochondrial function. You do want to think about that, of course. I guess that would be my answer to your question, is that everything is a network, everything should be balanced, and that it’s harder to do this in modern life because we have these poor lights, and poor environments, and stress and toxins. And our mitochondria are taking a big hit, as you know, which are the energy producing part of our cell. If your mitochondria aren’t working well, then everything else spirals from there. And obviously, the neurotransmitter ecosystem is a huge reflection of that too.
Dr. Gundry:
I think that’s the point. Yeah, each of these has a piece, but to call out one single piece is, I think, missing the bigger picture. Thank you.
Dr. Scott Sherr:
I’m happy to talk about that, because there are a lot of people out there that will focus on one thing. I am talking about GABA today as that one thing on some level, but the thing about GABA is that it’s such a ubiquitous molecule and neurotransmitter in the brain and what it does. And it reflects on mitochondrial function, it reflects on your sympathetic and parasympathetic balance.
Which I think is the big part here, Dr. Gundry, which is that so many of us, as I said, are living in that hustle culture, that we don’t realize that it’s actually when we relax and rest that we heal, we detox. If you’re somebody who likes to perform better, even at the gym, you make all of your muscle when you’re in parasympathetic mode. And so when your insulin level goes up after you eat, actually your GABA levels actually go up too. And so, then that’s why you feel better after a meal, is you feel more relaxed because that GABA is going up. And so, that GABA is going up trying to get you into that muscle anabolic state. If you’re in the sympathetic state all the time, you’re catabolic, you’re breaking things down. If you can get more into the parasympathetic state, you’re in the anabolic state. You’re building things up. As we get older, it’s much harder to stay in that anabolic state, if you’re always stressed all the time, especially.
Dr. Gundry:
Now, I know some people who are listening to us go, wait a minute, I’ve heard GABA. That reminds me of a drug that I’m taking called Gabapentin, that’s got the word GABA in it. You and I know that there are a great number of people who are given this drug for nerve pain, for neuropathy, and one of the side effects that everyone hopefully is warned about, is that you may get really sleepy or your brain may get kind of fuzzy. Are we talking about a similar compound here?
Dr. Scott Sherr:
It’s funny, like a lot of drugs in medicine that you well know, Dr. Gundry, they find these drugs and they work, but they have no idea why it works. That was Gabapentin for a long, long time. Gabapentin is a drug that works on the whole Gabaergic system, but it does it in a very unique and weird way on calcium uptake and presynaptic neurons, giving it a much higher risk for tolerance, withdrawal, and dependence over time. It’s a really hard drug to get off of, if people that are listening, it’s really difficult, but it’s affecting the GABA system. This GABA receptor, there’s different types, there’s GABA A and GABA B. Mostly we’re talking about GABA A receptors, and the GABA A receptors are the ones that have basically an area where GABA binds, and they have an area where what are called allosteric or other kinds of molecules can bind.
The most common thing that binds to GABA that most people, most of our patients, although maybe you and I are maybe different here, are alike in the capacity of alcohol. Alcohol is the most commonly used GABA binder. What it does is it binds to the GABA receptor, the GABA A receptor, then it increases the affinity for the actual GABA to bind. It binds very tightly to the receptor, and then it can deplete GABA very, very quickly. This is why you drink and then your sleep is trashed, you wake up two hours later. Because all that GABA is gone, and you have all that glutamate that’s in high proportion in comparison, so you have headaches, you have abdominal pain.
Dr. Scott Sherr:
That’s in high proportion in comparison. So you have headaches, you have abdominal pain, you feel like crap, you can’t move. And that’s all that glutamate toxicity that’s happening, at least significant portion.
So alcohol though, because it binds so tightly to the receptor, increases your risk of tolerance over time so you need more dependence on it because you have to have a certain amount or the system starts breaking down or starting to go into withdrawal.
And then of course, if you stop alcohol too quickly, if you’ve been drinking a lot, you can die because of instability in your system related to low amounts of GABA. So you have your benzodiazepines that do this too, your benzos, your Ativan, Xanax, Valium. That’s why they feel good. They feel good because it’s enhancing the GABA system. But again, binding so tightly to that receptor causing all these downstream things.
We know if you take one of those drugs, your risk for all cause mortality for anything goes up. And that’s the same thing with sleep drugs too like your Ambiens, Lunestas, all these things. But there are better ways to supplement the GABA system without using those drugs under most circumstances. And that’s what I’ve gotten really excited about from a diet, from a lifestyle perspective, but also supplementation. But certainly not taking a GABA supplement because that’s not going to work. And we can talk about that if you want.
Dr. Gundry:
Well, I think that’s a good place. But you mentioned something that probably went right over everybody’s head. What the heck is an orthosteric versus an allosteric modulation? Oh, come on, help us out here.
Dr. Scott Sherr:
Yeah, so this is a pretty easy concept. So imagine you have a receptor, okay, and then the receptor has a molecule that binds it. In this case it’s going to be GABA. So there’s going to be a location on the receptor where GABA is going to bind. That’s called, the fancy name for that is the orthosteric ligand. So basically it’s, an orthosteric ligand is the receptor binds to the molecule that it’s supposed to bind to. In this case it’s GABA. Okay.
Then you have what’s called an allosteric site, allosteric receptor sites. So if you have a GABA receptor, the allosteric sites are not where GABA would bind, but separate sites on the receptor that either can enhance or inhibit the amount of that particular molecule, in this case GABA, to bind to its actual receptor.
So you have allosteric modulators and you have orthosteric modulators. So most things that enhance or block the GABA receptor are going to be allosteric modulators. So in this case, we were talking about alcohol, benzodiazepines, sleep drugs, these are also all called PAMS, P-A-M-S, it’s positive allosteric modulators of the GABA receptor. So they basically increase the affinity for GABA to bind to where it would bind on the orthosteric ligand, so the orthosteric site of where GABA would bind.
So allosteric/orthosteric, I know that’s confusing, but in general, just know that they are things that bind to where GABA would bind, and there’s things that bind on other sites on that same receptor that increase or decrease how much GABA can bind.
Dr. Gundry:
Now, I interrupted you, you said that I just can’t swallow my GABA pill and accomplish much, how come?
Dr. Scott Sherr:
The problem is that GABA itself is too big of a molecule to get into the brain. The blood-brain barrier prevents many things that don’t have transporters to get through. And GABA is one of them. So if you’re taking a GABA supplement to try to help you relax, calm down, and it works for you, it could mean because you have what’s called a leaky brain or a leaky blood-brain barrier.
Now, this may sound scary, and honestly it kind of is, but it’s really common, especially if you have a leaky gut. These go together. If the gut is leaky, there’s more inflammation in the system, there’s going to be a leaky brain. And so if you’ve had a chronic infection for a while, if you have long COVID, for example, chronic EBV, all these kinds of things, very much associated with this leaky brain.
So from a clinical perspective, having a GABA supplement and having your patient respond to that is almost diagnostic for that blood-brain barrier not being intact. So in general, what you want to be doing is thinking about supporting that receptor by using compounds that are going to bind allosterically and orthosterically to it, but not using GABA on its own. And there’s some really cool compounds that do this.
Dr. Gundry:
On the same subject, a lot of mocktails claim are trying to duplicate the effects of alcohol on calming, inhibitions, on increasing your socialization, and they claim to be raising GABA levels with various herbs. So are they effective or are we going to go there next?
Dr. Scott Sherr:
So the thing with mostly what they’re using is something called kava. So kava has been around for a long time. It is a positive allosteric modulator of the GABA receptor. In short, it binds to the GABA receptor at a separate site where GABA would bind, and it enhances the amount of GABA to bind to the receptor. So kava has an alcohol-like effect. But what’s different from kava is that it doesn’t bind as tightly to the receptor. So as a result of that, it doesn’t cause the same potential to have withdrawal dependence and tolerance. Although all those things are possible if you are having a lot of kava. So you have to be careful.
So a lot of these other mocktails are using other kinds of herbs in there, passionflower, for example, is another one. Ashwagandha is another one, it also works on the GABA receptor.
Dr. Gundry:
Ashwagandha, yeah.
Dr. Scott Sherr:
So these are things that do enhance the GABA system. And when you’re using natural compounds, overall they’re going to be less binding tightly to the receptor. So they’re not going to have the same kind of addiction tolerance withdrawal profile. But it’s possible, valerian root, for example, is another one, valerian’s a commonly used sleep aid. It binds to the benzo receptor on the GABA receptor itself, on the benzo site. And so it works very well for a lot of people. It smells very terrible, as most people know, if they’ve smelled valerian root, it’s very pungent, but it does work as well.
But my concern here, Dr. Gundry, is that if you’re just working on supplementation on those other sites, but you’re not actually thinking about your GABA levels themselves and supporting them, you might not get into a place where you would with benzos and alcohol, but you can still get into a place where you’re depleting GABA even more over time.
So it’s really important, I think, to be thinking about the whole picture, making sure you can make enough GABA, convert it over to the glutamate. But there are other additional compounds that can actually work just like GABA in the brain and help you prevent any GABA depletion by taking some of these other compounds like kava or even CBD and CBG and other cannabinoids also bind to the GABA receptor.
Dr. Gundry:
All right, come on, don’t hold back, what are those compounds, we want to know?
Dr. Scott Sherr:
My favorite one, I led you on, I know, I know, I led you on, my favorite one is actually from the amanita muscaria mushroom, it’s also called the fly agaric mushroom. This is a psychedelic mushroom. Many of you have heard of it before. There’s some theories that Siberian winters in Russia where Christmas came about, they were using these mushrooms, drying them, and having psychedelic visions. Reindeers love to eat these mushrooms and trip actually. And there’s also Mario Brothers, Alice in Wonderland, a very popular mushroom.
There’s two compounds in the mushroom, one’s called ibotenic acid, that is neurotoxic, so you don’t want to have that one. But the other one is called agarin, and it is a long-acting agonist or enhances the GABA receptor by binding where GABA would bind. And its half-life is about six and a half hours, so it’s fantastic for sleep. And you use very small doses of this, a half a milligram, a milligram, maybe up to two milligrams at most. And in these very small amounts, if you combine it with something that’s also enhancing the GABA receptor by binding to those other sites, it’s this beautiful one-two synergistic combination. So you can use kava with it, you could use another one called magnolia bark or [inaudible 00:23:31] or DDHP, which is a more strong version of that, which is another allosteric modulator positive of the GABA receptor.
So our favorite by far is agarin because you have this cool story of course, but it also is fantastically effective. And it’s really great to use in a sleep aid because in a sleep aid, you have the long-acting need for having GABA around, and that GABA support can go a long way from people not only falling asleep pretty easily. But the hardest thing I think for people oftentimes is when they wake up in the middle of the night and they can’t …
Dr. Scott Sherr:
… thing I think for people oftentimes is when they wake up in the middle of the night and they can’t go back to bed, mind starts racing. So, having the agrin on board is really a great way to support that GABA system, alongside of having… Obviously trying to optimize your sleep in other ways too, but optimizing your GABA system in correspondence.
And the other one that we like other than agrin is something called B3 GABA, nicotinoyl-GABA. This particular compound is a GABA that’s attached to a vitamin B3. And what’s cool about this one is that B3 has a transporter that gets across the brain, and so B3 kind of takes GABA with it, even if the blood brain barrier is intact, which hopefully it is. And then in the brain you have a little bit of B3 and you have a little bit of GABA, and so the B3 is actually mildly activating, because it turns into niacin, NAD, and then you have obviously the GABA, which can bind to where on the receptor where GABA would bind. And so you have this relaxation, but you don’t have the fatigue or sedation that often comes with taking supplements or medications that work on the GABA system.
So, that’s another cool one that I like to use, that we like to use.
Dr. Gundry:
Now, if we take these mushroom compounds, will the DEA be knocking on our door?
Dr. Scott Sherr:
So no. Agrin is not scheduled or anything, it’s not going to cause a psychedelic experience at low doses. The only state in the United States that doesn’t allow you to have the amanita mushroom is Louisiana, interestingly enough. But the mushroom itself people do use in various concoctions and potions and tinctures and things, but it will have some of that ibotenic acid in it, so I usually don’t recommend using it for that reason. I try to avoid things that are neurotoxic in general, if I can.
Dr. Gundry:
Good idea.
Dr. Scott Sherr:
But so no, this is not like your magic mushrooms or your psilocybin mushrooms or LSD, it’s not going to cause you to trip. It’s just going to give you that enhanced GABA activity in a more holistic way. So again, the key I think is to really replete the GABA on multiple fronts. So, looking at it from a diet, a lifestyle perspective, downregulating your nervous system, looking at your micronutrients, and then supplementing it in a way that you’re not only just enhancing GABA to bind, but you’re also preventing the depletion of it at the same time.
Dr. Gundry:
Perfect, good summary. We have an audience question. I’m going to let you take this first. I know what you’re going to say, but I want… Kristy813 on Instagram writes, “What should I look for in a GABA supplement? Or better yet, what do I avoid on the label? Is there a dosage level I should look for?”
Dr. Scott Sherr:
Well, this podcast is a summary, or I guess the long summary is the whole podcast, right? The short summary would be that you don’t want to take GABA supplements. GABA supplements directly do not work. If you do take them and they work for you, it’s probably because you need some help with optimizing your gut and optimizing your blood-brain barrier, and you can do that work with a practitioner. There’s many that can help you here just optimizing your gut, optimizing your brain will come in correspondence.
And so, many people are taking these combination sleep supplements at night. It might have some GABA, it might have some L-theanine, which is another allosteric modulator, the GABA receptor. In general, I have watch out for anything that would have fillers and sugar and all that other crap that people will find in some of these sleep supplements. But what I like to do is think about this, what we call an obligate pair strategy.
A strategy where you’re enhancing the GABA system by having something bind to those allosteric sites. So, kava, CBD, CBG, honokiol, for example, those are some options. And then having something that binds directly to where GABA would bind, so you’re not preventing, or you’re not causing, I should say, any GABA depletion. So, this would be something like agarin or something like nicotinoyl-GABA, for example. So those are the ones… That’s the comprehensive strategy where you take one compound that binds to where GABA would bind and one compound that binds to the other site, and you combine them together in a way that really enhances the GABA system overall.
So, from a dosing perspective there may be some… Everybody’s going to be different here, so I usually recommend they’re starting off at low doses of these kinds of things and synergizing them together, and there’s various products out there that might be able to help you. And finding your dose. I always like to say, go low and slow with these kinds of things. Don’t go to the highest dose initially. You start off low and just be careful with your dosing and understand that it might be a big spectrum for people.
And then once your GABA system starts coming back online, you’re working on your parasympathetic nervous system. So you’re doing breath work, doing yoga, you’re exercising, you’re eating better glutamine containing foods, you’ve optimized your gut, your magnesium levels are better, and hopefully you can come and use less of these products, but you can use them on demand.
So, if you’re in a stressful situation, you’re getting on an airplane, or you’re about to give a talk, or you’re about to get a surgery, these are kinds of different reasons why it might be nice to take the GABA and optimize it using a supplement strategy like this.
Dr. Gundry:
Well, that’s great, yeah. That’s why I wanted to have you on the program, because I think this is one place where it’s GABA and what it’s doing and how to influence has been lost in the overall discussion of all these neurotransmitters. And that was a great summary, and I appreciate you taking the time to come on the podcast.
Dr. Scott Sherr:
My pleasure, Dr. Gundry, it’s been a pleasure to be with you.
Dr. Gundry:
Where can people find you and your work?
Dr. Scott Sherr:
So you can find me, my website is Drscottsherr.com, D-R-S-C-O-T-T-S-H-E-R-R. If you’re interested in some of my social media and presence there, I’m at @DrScottScherr. I also have a company called Troscriptions, T-R-O and then prescriptions at the end there, Troscriptions, and you can find that at Troscriptions.com as well as @Troscriptions on Instagram. And we have some products that are involved in the work that we were just describing.
My focus is as a clinician, this is what I do in clinical practice, and my hope is to create more of an understanding and more visibility on GABA deficiency as running the show in so many of these conditions that we’re treating with band-aids in clinical practice. So yeah, Troscriptions.com, Drscottscherr.com, and then the corresponding Instagram, social media handles, YouTube, those kinds of things. You can find us there.
Dr. Gundry:
Great. And thank you very much once again and hope to see you soon.
Now it’s time for the review of the week. This one’s from @KarenCampbell8248 on YouTube, “Great suggestions in your video. In the toilet bowl section, you show a picture of Seventh Generation toilet bowl cleaner that appears to have all botanical ingredients. I have used it for years in researched it as a safe choice.”
Well, that’s very observant of you, KarenCampbell8248. Seventh Generation makes some great products and really looks out for you and the environment. Thanks for reviewing.
Speaker 1:
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